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See also Chromium and magnesium effects on blood sugar.
James Sloane
There is a common misconception that all forms of sugar diabetes result from pancreatic failure or damage. This is not true except in cases of type 2 diabetes. Neither type 2 diabetes that accounts for 95% of the cases of sugar diabetes and gestational diabetes do not result from pancreatic failure or damage.
In type 2 diabetes there is a closing of insulin receptors. Therefore, in the early stages of type 2 diabetes the pancreas produces plenty of insulin, but the insulin cannot be utilized properly by the cells do to the lack of open insulin receptors.
Normally insulin latches on to insulin receptors on the cells sensitizing the cells to uptake glucose. {If those} receptors are closed or blocked they cannot be sensitized by the insulin and therefore blood sugar levels remain higher than normal.
Elevated blood glucose leads to excess urine secretion, which is the actual definition of diabetes. This is due to the glucose drawing water through the kidneys as excess glucose is being eliminated through the kidneys. Other side effects include fatigue, excessive hunger, excessive thirst, blurred vision, tingling or pain in the feet, etc.
In the long run the elevated glucose raises triglyceride levels, leads to diabetic cataracts and demyelinates the nerves leading to neuropathy.
The real dangerous side effects though such as retinopathy, kidney failure, gangrene and a lot of the heart disease is not the result of the elevated blood sugar, but rather the elevated insulin. In short insulin levels creep up from the insulin resistance leading to the blood sugar remaining high. So more insulin is secreted leading to excess insulin levels, which leads to strong constriction of the blood vessels rupturing micro-blood vessels leading to retinopathy, kidney failure and gangrene. The resultant high blood pressure leads to arterial damage producing inflammation that in turn leads to heart disease.
Therefore, the bottom line is it is very important to get the insulin receptors back open. If the person is overweight losing some weight can help since excess body fat decreases insulin sensitivity. If associated with polycystic ovarian syndrome (PCOS) then the main goal should be to get the hormones back in to balance.
More commonly though the insulin resistance is due to a lack of chromium and/or magnesium. See: {No, they’re dead links.}
http://medcapsules.com/forum/showthread.php?tid=3160
http://medcapsules.com/forum/showthread.php?tid=3159
The best source of chromium is chromium polynicotinate. Chromium polynicotinate is 300 times more effective than chromium picolinate and the same cost. Recommended dose is 200mcg 3 to 4 times daily.
The best form of magnesium is magnesium malate. Do not use magnesium oxide which is poorly absorbed, dangerously neutralizes stomach acid and mainly works as a laxative. Magnesium malate is better absorbed, does not neutralize stomach acid and is great for increasing levels of adenosine triphosphate (ATP), which is what fuels cells and helps them to work properly.
Magnesium also helps with insulin production and dilates the blood vessels by acting like a natural calcium channel blocker preventing insulin damage. Because it works by antagonizing calcium it is best taken away from calcium in this case. Recommended dose of magnesium (measured as elemental magnesium) is 300-400mg 2-3 times daily. Start out with lower doses and build a tolerance slowly since magnesium can still be laxative in other forms.